12th Edition of International Conference on Neurology and Brain Disorders
October 20-22, 2025
Neuroinflammation and Brain Disorders
Neuroinflammation is increasingly recognized as a key factor in the development and progression of a variety of brain disorders. Chronic neuroinflammation contributes to the breakdown of the blood-brain barrier and the accumulation of toxic proteins, leading to neuronal injury and loss. In the context of neuroinflammation and brain disorders, conditions such as Alzheimer's, Parkinson's, and autoimmune diseases like multiple sclerosis are significantly impacted by this inflammatory response. While initially protective, neuroinflammation can become maladaptive, further promoting brain damage. Advances in research are revealing the role of immune cells, such as microglia and T-cells, in driving neuroinflammation and how this process is directly linked to the progression of neuroinflammation and brain disorders. New therapies targeting neuroinflammatory pathways, including anti-inflammatory cytokines and microglial inhibitors, offer potential treatments to mitigate these effects and slow the progression of brain disorders.
Ken Ware
NeuroPhysics Therapy Institute and Research Centre, Australia
Joe Sam Robinson
Mercer University, United States
Robert B Slocum
University of Kentucky HealthCare, United States
Thomas J Webster
Interstellar Therapeutics, United States
Roger H Coletti
Interventional Health, PA, United States
Stephen Grossberg
Boston University, United States
George Diaz
Memorial Healthcare Systems, United StatesSubmit your abstract Today
Title : A case of vile vindictive primary CNS vasculitis
George Diaz, Memorial Healthcare Systems, United States
Title : Novel important cellular responses, signaling mechanisms and therapeutic options in vascular dementia
Yong Xiao Wang, Albany Medical College, United States
Title : The role of beliefs, perception, and behavioural patterns in the evolution of psychophysical disorders
Ken Ware, NeuroPhysics Therapy Institute and Research Centre, Australia
Title : A multiscale systems biology framework integrating ODE-based kinetics and MD-derived structural affinities to model mBDNF–proBDNF-mediated bifurcation dynamics in CNS neurotrophin signaling
Krishna Moorjani, Boston University, United States
Title : A multiscale systems biology framework integrating ODE-based kinetics and MD-derived structural affinities to model mBDNF–proBDNF-mediated bifurcation dynamics in CNS neurotrophin signaling
Abhay Murthy, Boston University, United States
Title : A multiscale systems biology framework integrating ODE-based kinetics and MD-derived structural affinities to model mBDNF–proBDNF-mediated bifurcation dynamics in CNS neurotrophin signalling
Ethan Liu, Boston University, United States