Title : Neurogenetic alternations due to intensive exposure to mobile phone radiations
Abstract:
Excessive implications of mobile phone radiations and nanotechnology with prolong duration can lead to several neurological disparities; at an optimum state including epigenetic alterations, NGF/BDNF problems as well as misinterpret secretions of acetylcholine (PSNS), norepinephrine (SNS), dopamine and glutamate levels. Since after proliferation neurons of basal ganglia (striatum), nucleus basalis and hippocampus (regions of mid brain) are in a migratory and functional state of differentiation, they show immersive physiological changes with due time. FMRI and TMS study furthermore sheds light on the behaviour of individuals with respect to neural impairments after given profound amount of intensity and radiations in various research studies. This leads to easy alternation of genetic material in brain, just like methylation and acetylation (switch on and off receptors of DNA and histone proteins) does while being hefty on drugs or alcoholic intake. Apart from the neural cortex, it as well specifically harms the visual cortex of our brain, incorporating both regions of inferior temporal cortex and the inferior parietal ones (running parallel to A1-A2 regions). Though eyesight is affected by it, regions of MT, MST and V5 (V5/MT) at times can as well lose control over significant functions under this process. Not just this, but facial recognition (V3); which is a premiere category of study for neurologists, might showcase an elevated level of disfunction in such scenarios; thereby alternating associated aging problems like Alzheimer’s etc. Following the same, somatosensory cortex and underlying pre central gyri might show impressive reactions to stream of binding problems and stiffness over vestibular organs (otoliths) as a primary result. Therefore, my research paper will focus on all such biological and psychological parameters associated along with mobile phone radiations and nanotechnology.
Key words: striatum, nucleus basalis, hippocampus, V5/MT, binding problems, NGF/BDNF, epigenetics
